primary monoclonal rabbit anti-hent1 (Spring Bioscience)
90
Structured Review
Spring Bioscience
primary monoclonal rabbit anti-hent1
Primary Monoclonal Rabbit Anti Hent1, supplied by Spring Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+monoclonal+rabbit+anti-hent1/primary+monoclonal+rabbit+anti+hent1/pm25869606-121-52-59
Average 90 stars, based on 1 article reviews
Primary Monoclonal Rabbit Anti Hent1, supplied by Spring Bioscience, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+monoclonal+rabbit+anti-hent1/primary+monoclonal+rabbit+anti+hent1/pm25869606-121-52-59
Average 90 stars, based on 1 article reviews
primary monoclonal rabbit anti-hent1 - by Bioz Stars,
2026-10
90/100 stars
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Cell Culture:Article Title: Insulin requires normal expression and signaling of insulin receptor A to reverse gestational diabetes-reduced adenosine transport in human umbilical vein endothelium. Article Snippet: Reduced adenosine uptake via human equilibrative nucleoside transporter 1 (hENT1) in human umbilical vein endothelial cells (HUVECs) from gestational diabetes mellitus (GDM) is reversed by insulin by restoring hENT1 expression.. Insulin receptors A (IR-A) and B (IR-B) are expressed in HUVECs, and GDM results in higher IR-A mRNA expression vs. cells from normal pregnancies.. We studied whether the reversal of GDM effects on transport by insulin depends on restoration of IR-A expression.We specifically measured hENT1 expression [mRNA, protein abundance, SLC29A1 (for hENT1) promoter activity] and activity (adenosine transport kinetics) and the role of IR-A/IR-B expression and signaling [total and phosphorylated 42 and 44 kDa mitogen-activated protein kinases (p44/42) and Akt] in IR-A, IR-B, and IR-A/B knockdown HUVECs from normal (n = 33) or GDM (n = 33) pregnancies. Article Title: Human supraphysiological gestational weight gain and fetoplacental vascular dysfunction. Article Snippet: 19 In brief, cells were cultured in the absence or presence of 10 μg/mL CPZ as for adenosine transport assays and then fixed (4% paraformaldehyde, 5 min), permeabilized (0.2% Triton X-100, 10 min), and blocked (5% BSA, 0.02% Triton X-100, 30 min). hENT1 was immunolocalized following incubating (overnight at 4°C) with primary monoclonal rabbit anti-hENT1 (1:30) (catalog number: Incubation:Article Title: Insulin requires normal expression and signaling of insulin receptor A to reverse gestational diabetes-reduced adenosine transport in human umbilical vein endothelium. Article Snippet: Reduced adenosine uptake via human equilibrative nucleoside transporter 1 (hENT1) in human umbilical vein endothelial cells (HUVECs) from gestational diabetes mellitus (GDM) is reversed by insulin by restoring hENT1 expression.. Insulin receptors A (IR-A) and B (IR-B) are expressed in HUVECs, and GDM results in higher IR-A mRNA expression vs. cells from normal pregnancies.. We studied whether the reversal of GDM effects on transport by insulin depends on restoration of IR-A expression.We specifically measured hENT1 expression [mRNA, protein abundance, SLC29A1 (for hENT1) promoter activity] and activity (adenosine transport kinetics) and the role of IR-A/IR-B expression and signaling [total and phosphorylated 42 and 44 kDa mitogen-activated protein kinases (p44/42) and Akt] in IR-A, IR-B, and IR-A/B knockdown HUVECs from normal (n = 33) or GDM (n = 33) pregnancies. Article Title: Human supraphysiological gestational weight gain and fetoplacental vascular dysfunction. Article Snippet: 19 In brief, cells were cultured in the absence or presence of 10 μg/mL CPZ as for adenosine transport assays and then fixed (4% paraformaldehyde, 5 min), permeabilized (0.2% Triton X-100, 10 min), and blocked (5% BSA, 0.02% Triton X-100, 30 min). hENT1 was immunolocalized following incubating (overnight at 4°C) with primary monoclonal rabbit anti-hENT1 (1:30) (catalog number: |